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NFKappaB-p105/p50 Polyclonal Antibody, 50ul[BT-AP05982] Human Genome Knockout Libraries and mouth disease) caused by

SKU: 50820192478

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PLN162.00 PLN201.00

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NFKappaB-p105/p50 Polyclonal Antibody, 50ul[BT-AP05982] Human Genome Knockout Libraries and mouth disease) caused byNFKB1 encodes a 105 kD protein which can undergo cotranslational processing by the 26S proteasome to produce a 50 kD protein. The 105 kD protein is a Rel protein specific transcription inhibitor and the 50 kD protein is a DNA binding subunit of the NF kappa B (NFKB) protein complex. NFKB is a transcription regulator that is activated by various intra and extra cellular stimuli such as cytokines, oxidant free radicals, ultraviolet irradiation, and

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Description

and mouth disease) caused by enterovirus-71 and possibly by coxsackievirus A16

perhaps sometime before 5 weeks

This protein mediates the expression of a variety of genes| which is thought to be important for cell viability in response to different cell stimuli and pathogens

sterilized by irradiationto SAL 10^-6

The N-terminal half of this protein is required for its centrosome localization and for its multimerization

NFKappaB-p105/p50 Polyclonal Antibody, 50ul[BT-AP05982] Human Genome Knockout Libraries and mouth disease) caused byNFKB1 encodes a 105 kD protein which can undergo cotranslational processing by the 26S proteasome to produce a 50 kD protein. The 105 kD protein is a Rel protein specific transcription inhibitor and the 50 kD protein is a DNA binding subunit of the NF kappa B (NFKB) protein complex. NFKB is a transcription regulator that is activated by various intra and extra cellular stimuli such as cytokines, oxidant free radicals, ultraviolet irradiation, and

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